GHK-Cu – 200MG

GHK-Cu Peptide

GHK-Cu is a naturally-occurring complex molecule made of a tripeptide: GHK (glycine, histidine, and lysine amino acids) linked to a copper ion. The more stabilized copper ion then delivers GHK to cells. Studies show that GHK-Cu may be able to aid in wound-healing, tissue repair, and immune response. Which is probably because it may enhance collagen, elastin and glycosaminoglycans, essential intercellular matrix matters.

Besides its intercellular matrix potential, GHK-Cu may also be an anti-oxidant and anti-inflammatory, protecting cells from damage caused by haloalkanes. But the body's levels of GHK-Cu drop as we get older, which can help explain some of the age-related decline in recovery and immunity.

Photodamage in tissues, such as the skin, is directly related to the production of reactive oxygen species (ROS), reactive nitrogen species (RNS) and carbonyl compounds (RCS), which can induce damage to proteins, DNA and lipids. Research has demonstrated that, through scavenging actions on one or more types of free radicals (especially 4-hydroxynoneal, acrolein, and other RCS including malondialdehyde), the amino acid GHK-Cu in GHK product may have significant anti-radical effects. It also has the potential to stop glycation of proteins. In addition, it has been speculated by researchers that GHK-Cu may inhibit iron release from ferritin. Ferritin can act as an iron-protein complex that catalyzes lipid peroxidation, while GHK-Cu possibly helps to prevent the creation of these iron complexes within the damaged tissue and reduces inflammation. Scientists demonstrated in one study of injured tissues that GHK-Cu aided in lowering the oxidation and produced an 87% reduction in iron release.

GHK-Cu also inhibits the generation of reactive oxygen species and pro-inflammatory cytokines with a concurrent enhancement of antioxidant enzyme activity. It comes from an animal experiment that investigated whether GHK-Cu might protect against lipopolysaccharide-induced lung inflammation in mice. This peptide was proposed to inhibit NF-κB and p38 MAPK signaling pathways with inflammation. This might ultimately result in decreased recruitment and infiltration of inflammatory cells into the lungs of injured mice and correcting reduced levels of TNF-1 and IL-6 production at least bilaterally.

Also, GHK-Cu has also been suggested to decrease oxidative stress due to mechanisms related to smoking. Studies of cultured alveolar epithelial cells show that GHK normalizes expression of the Nrf2 protein and lowers stress induced levels of reactive oxygen species but fails to do so for COPD fibroblasts.

Similarly, other studies have also indicated another antioxidative action of aminopeptides that might be ascribed to the amino-acid sequence of GHK-Cu on ROS, such as hydroxyl radicals, an effect which could even be stronger than that of generic antioxidative peptides.

THE CONTENT ON THIS WEBSITE IS STRICTLY INFORMATIONAL. IT IS NOT INTENDED TO BE TAKEN AS MEDICAL ADVICE OR AS A SCIENTIFIC PUBLICATION.

None of the products have been evaluated by The Food and Drug Administration, and are not for medical use. Their use in human or animal subjects is strictly prohibited by law, and they are available here only for in-vitro research purposes, by licensed professionals. By purchasing from this site, you agree to comply with our Terms and Conditions.